Control of immune function, longevity and the genetic susceptibility
to certain diseases only limited DNA molecule fragments. Such as the
longevity and short-lived NZB NZW chromosome 17 in mice is H-2 region
genes of small differences.
Bacteria can live enzymes from their mother split the algebra
determined. In its lifetime, there are two different RAS1 and RAS2.
Make life shorter RAS1, RAS2 to life extension.
Mammalian genetic research found that H-2 area in mice at least 6 loci
and longevity is related to immune system changes in internal and
external environment is the first defense line of defense, with the
early life of the antibody response shows that the low immune response
higher than the response in mice live longer, study found that human
HLA locus, APOE and ACE area and lifespan.
Recently, the immune genetic studies indicate that HLA-DRW9 hundred
years old middle-aged low frequency of gene, HLA-DR1 frequency and
more, so that DRW9 are risk factors, DR1 is conducive to human
longevity genes. Furthermore is the telomere (telomere) on a close
relationship with aging. Telomere length was shortened with the
pull-aging, telomerase is able to eliminate the progressive
shortening, which makes it possible at the genetic level anti- aging,
in recent years, many related genes, such as SIR, P16 so closely with
the life of cells related.
Studies on the life span of genes yet to be thorough, despite the
family of the world have a number of longevity, but by no means
"fatalism." Environmental factors, metabolic rate, stress ability and
lifestyle conditions on the life of the day after tomorrow is in the
affirmative.
